Characteristics of Slow Pathway Conduction After Successful AVNRT Ablation
Identifieur interne : 002867 ( Main/Exploration ); précédent : 002866; suivant : 002868Characteristics of Slow Pathway Conduction After Successful AVNRT Ablation
Auteurs : Emoke Posan [Canada] ; Lorne J. Gula [Canada] ; Allan C. Skanes [Canada] ; Andrew D. Krahn [Canada] ; Raymond Yee [Canada] ; Basilios Petrellis [Canada] ; Damian P. Redfearn [Canada] ; Uwais Mohamed [Canada] ; Paul A. Gould [Canada] ; George J. Klein [Canada]Source :
- Journal of Cardiovascular Electrophysiology [ 1045-3873 ] ; 2006-08.
English descriptors
- KwdEn :
Abstract
Background: AV node slow pathway conduction can persist following successful ablation for AV node reentrant tachycardia (AVNRT). We hypothesized that careful examination of AV nodal conduction curves before and after effective AVNRT ablation in patients with persistent slow pathway conduction could shed light on this apparent paradox. Methods and Results: Thirty patients (age 40.9 ± 14.3; 8 male) were included. AV node function curves were created based on pre‐ and postablation atrial extrastimulus testing. Analysis of slow pathway function curves demonstrated significant decrease in AH for any given coupling interval after ablation (mean difference –68.1 [–94.5, –41.7] P < 0.001), graphically indicated by downward displacement of the curve. In addition, mean slow pathway effective refractory period (ERP) increased from 247.9 ± 36.1 msec to 288.6 ± 56.0 msec (P < 0.001); mean maximum AH interval decreased from 361.3 ± 114.2 msec to 306.9 ± 65.2 msec (P = 0.013); mean difference in minimum and maximum AH interval during slow pathway conduction decreased (from 94.5 ± 75.8 msec to 59.6 ± 46.2 msec (P = 0.016). Finally, mean difference between the fast and slow pathway effective refractory periods, the span of coupling intervals over which slow pathway conduction occurred, decreased (from 113.9 ± 61.4 msec to 63.2 ± 41.5 msec, P = 0.001). Conclusions: Ablation, which successfully eliminates inducible and spontaneous AVNRT in the presence of persistent slow pathway conduction, is associated with significantly altered slow pathway conduction characteristics, indicating the presence of a damaged or different slow pathway after ablation, incapable of sustaining tachycardia.
Url:
DOI: 10.1111/j.1540-8167.2006.00492.x
Affiliations:
Links toward previous steps (curation, corpus...)
- to stream Istex, to step Corpus: 000756
- to stream Istex, to step Curation: 000756
- to stream Istex, to step Checkpoint: 000C10
- to stream Main, to step Merge: 002B54
- to stream Main, to step Curation: 002867
Le document en format XML
<record><TEI wicri:istexFullTextTei="biblStruct"><teiHeader><fileDesc><titleStmt><title xml:lang="en">Characteristics of Slow Pathway Conduction After Successful AVNRT Ablation</title><author><name sortKey="Posan, Emoke" sort="Posan, Emoke" uniqKey="Posan E" first="Emoke" last="Posan">Emoke Posan</name></author><author><name sortKey="Gula, Lorne J" sort="Gula, Lorne J" uniqKey="Gula L" first="Lorne J." last="Gula">Lorne J. Gula</name></author><author><name sortKey="Skanes, Allan C" sort="Skanes, Allan C" uniqKey="Skanes A" first="Allan C." last="Skanes">Allan C. Skanes</name></author><author><name sortKey="Krahn, Andrew D" sort="Krahn, Andrew D" uniqKey="Krahn A" first="Andrew D." last="Krahn">Andrew D. Krahn</name></author><author><name sortKey="Yee, Raymond" sort="Yee, Raymond" uniqKey="Yee R" first="Raymond" last="Yee">Raymond Yee</name></author><author><name sortKey="Petrellis, Basilios" sort="Petrellis, Basilios" uniqKey="Petrellis B" first="Basilios" last="Petrellis">Basilios Petrellis</name></author><author><name sortKey="Redfearn, Damian P" sort="Redfearn, Damian P" uniqKey="Redfearn D" first="Damian P." last="Redfearn">Damian P. Redfearn</name></author><author><name sortKey="Mohamed, Uwais" sort="Mohamed, Uwais" uniqKey="Mohamed U" first="Uwais" last="Mohamed">Uwais Mohamed</name></author><author><name sortKey="Gould, Paul A" sort="Gould, Paul A" uniqKey="Gould P" first="Paul A." last="Gould">Paul A. Gould</name></author><author><name sortKey="Klein, George J" sort="Klein, George J" uniqKey="Klein G" first="George J." last="Klein">George J. Klein</name></author></titleStmt><publicationStmt><idno type="wicri:source">ISTEX</idno><idno type="RBID">ISTEX:B48FB2053E9197FF284325910055B400156E0503</idno><date when="2006" year="2006">2006</date><idno type="doi">10.1111/j.1540-8167.2006.00492.x</idno><idno type="url">https://api-v5.istex.fr/document/B48FB2053E9197FF284325910055B400156E0503/fulltext/pdf</idno><idno type="wicri:Area/Istex/Corpus">000756</idno><idno type="wicri:explorRef" wicri:stream="Istex" wicri:step="Corpus" wicri:corpus="ISTEX">000756</idno><idno type="wicri:Area/Istex/Curation">000756</idno><idno type="wicri:Area/Istex/Checkpoint">000C10</idno><idno type="wicri:explorRef" wicri:stream="Istex" wicri:step="Checkpoint">000C10</idno><idno type="wicri:doubleKey">1045-3873:2006:Posan E:characteristics:of:slow</idno><idno type="wicri:Area/Main/Merge">002B54</idno><idno type="wicri:Area/Main/Curation">002867</idno><idno type="wicri:Area/Main/Exploration">002867</idno></publicationStmt><sourceDesc><biblStruct><analytic><title level="a" type="main" xml:lang="en">Characteristics of Slow Pathway Conduction After Successful AVNRT Ablation</title><author><name sortKey="Posan, Emoke" sort="Posan, Emoke" uniqKey="Posan E" first="Emoke" last="Posan">Emoke Posan</name><affiliation wicri:level="1"><country xml:lang="fr">Canada</country><wicri:regionArea>Division of Cardiology, Department of Medicine, London Health Sciences Centre, London, Ontario</wicri:regionArea><wicri:noRegion>Ontario</wicri:noRegion></affiliation></author><author><name sortKey="Gula, Lorne J" sort="Gula, Lorne J" uniqKey="Gula L" first="Lorne J." last="Gula">Lorne J. Gula</name><affiliation wicri:level="1"><country xml:lang="fr">Canada</country><wicri:regionArea>Division of Cardiology, Department of Medicine, London Health Sciences Centre, London, Ontario</wicri:regionArea><wicri:noRegion>Ontario</wicri:noRegion></affiliation></author><author><name sortKey="Skanes, Allan C" sort="Skanes, Allan C" uniqKey="Skanes A" first="Allan C." last="Skanes">Allan C. Skanes</name><affiliation wicri:level="1"><country xml:lang="fr">Canada</country><wicri:regionArea>Division of Cardiology, Department of Medicine, London Health Sciences Centre, London, Ontario</wicri:regionArea><wicri:noRegion>Ontario</wicri:noRegion></affiliation></author><author><name sortKey="Krahn, Andrew D" sort="Krahn, Andrew D" uniqKey="Krahn A" first="Andrew D." last="Krahn">Andrew D. Krahn</name><affiliation wicri:level="1"><country xml:lang="fr">Canada</country><wicri:regionArea>Division of Cardiology, Department of Medicine, London Health Sciences Centre, London, Ontario</wicri:regionArea><wicri:noRegion>Ontario</wicri:noRegion></affiliation></author><author><name sortKey="Yee, Raymond" sort="Yee, Raymond" uniqKey="Yee R" first="Raymond" last="Yee">Raymond Yee</name><affiliation wicri:level="1"><country xml:lang="fr">Canada</country><wicri:regionArea>Division of Cardiology, Department of Medicine, London Health Sciences Centre, London, Ontario</wicri:regionArea><wicri:noRegion>Ontario</wicri:noRegion></affiliation></author><author><name sortKey="Petrellis, Basilios" sort="Petrellis, Basilios" uniqKey="Petrellis B" first="Basilios" last="Petrellis">Basilios Petrellis</name><affiliation wicri:level="1"><country xml:lang="fr">Canada</country><wicri:regionArea>Division of Cardiology, Department of Medicine, London Health Sciences Centre, London, Ontario</wicri:regionArea><wicri:noRegion>Ontario</wicri:noRegion></affiliation></author><author><name sortKey="Redfearn, Damian P" sort="Redfearn, Damian P" uniqKey="Redfearn D" first="Damian P." last="Redfearn">Damian P. Redfearn</name><affiliation wicri:level="1"><country xml:lang="fr">Canada</country><wicri:regionArea>Division of Cardiology, Department of Medicine, London Health Sciences Centre, London, Ontario</wicri:regionArea><wicri:noRegion>Ontario</wicri:noRegion></affiliation></author><author><name sortKey="Mohamed, Uwais" sort="Mohamed, Uwais" uniqKey="Mohamed U" first="Uwais" last="Mohamed">Uwais Mohamed</name><affiliation wicri:level="1"><country xml:lang="fr">Canada</country><wicri:regionArea>Division of Cardiology, Department of Medicine, London Health Sciences Centre, London, Ontario</wicri:regionArea><wicri:noRegion>Ontario</wicri:noRegion></affiliation></author><author><name sortKey="Gould, Paul A" sort="Gould, Paul A" uniqKey="Gould P" first="Paul A." last="Gould">Paul A. Gould</name><affiliation wicri:level="1"><country xml:lang="fr">Canada</country><wicri:regionArea>Division of Cardiology, Department of Medicine, London Health Sciences Centre, London, Ontario</wicri:regionArea><wicri:noRegion>Ontario</wicri:noRegion></affiliation></author><author><name sortKey="Klein, George J" sort="Klein, George J" uniqKey="Klein G" first="George J." last="Klein">George J. Klein</name><affiliation wicri:level="1"><country xml:lang="fr">Canada</country><wicri:regionArea>Division of Cardiology, Department of Medicine, London Health Sciences Centre, London, Ontario</wicri:regionArea><wicri:noRegion>Ontario</wicri:noRegion></affiliation></author></analytic><monogr></monogr><series><title level="j">Journal of Cardiovascular Electrophysiology</title><idno type="ISSN">1045-3873</idno><idno type="eISSN">1540-8167</idno><imprint><publisher>Blackwell Publishing Inc</publisher><pubPlace>Malden, USA</pubPlace><date type="published" when="2006-08">2006-08</date><biblScope unit="volume">17</biblScope><biblScope unit="issue">8</biblScope><biblScope unit="page" from="847">847</biblScope><biblScope unit="page" to="851">851</biblScope></imprint><idno type="ISSN">1045-3873</idno></series><idno type="istex">B48FB2053E9197FF284325910055B400156E0503</idno><idno type="DOI">10.1111/j.1540-8167.2006.00492.x</idno><idno type="ArticleID">JCE492</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">1045-3873</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>AV node</term><term>AVN reentry</term><term>AVNRT</term><term>catheter ablation</term><term>conduction</term><term>electrophysiology</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Background: AV node slow pathway conduction can persist following successful ablation for AV node reentrant tachycardia (AVNRT). We hypothesized that careful examination of AV nodal conduction curves before and after effective AVNRT ablation in patients with persistent slow pathway conduction could shed light on this apparent paradox. Methods and Results: Thirty patients (age 40.9 ± 14.3; 8 male) were included. AV node function curves were created based on pre‐ and postablation atrial extrastimulus testing. Analysis of slow pathway function curves demonstrated significant decrease in AH for any given coupling interval after ablation (mean difference –68.1 [–94.5, –41.7] P < 0.001), graphically indicated by downward displacement of the curve. In addition, mean slow pathway effective refractory period (ERP) increased from 247.9 ± 36.1 msec to 288.6 ± 56.0 msec (P < 0.001); mean maximum AH interval decreased from 361.3 ± 114.2 msec to 306.9 ± 65.2 msec (P = 0.013); mean difference in minimum and maximum AH interval during slow pathway conduction decreased (from 94.5 ± 75.8 msec to 59.6 ± 46.2 msec (P = 0.016). Finally, mean difference between the fast and slow pathway effective refractory periods, the span of coupling intervals over which slow pathway conduction occurred, decreased (from 113.9 ± 61.4 msec to 63.2 ± 41.5 msec, P = 0.001). Conclusions: Ablation, which successfully eliminates inducible and spontaneous AVNRT in the presence of persistent slow pathway conduction, is associated with significantly altered slow pathway conduction characteristics, indicating the presence of a damaged or different slow pathway after ablation, incapable of sustaining tachycardia.</div></front></TEI><affiliations><list><country><li>Canada</li></country></list><tree><country name="Canada"><noRegion><name sortKey="Posan, Emoke" sort="Posan, Emoke" uniqKey="Posan E" first="Emoke" last="Posan">Emoke Posan</name></noRegion><name sortKey="Gould, Paul A" sort="Gould, Paul A" uniqKey="Gould P" first="Paul A." last="Gould">Paul A. Gould</name><name sortKey="Gula, Lorne J" sort="Gula, Lorne J" uniqKey="Gula L" first="Lorne J." last="Gula">Lorne J. Gula</name><name sortKey="Klein, George J" sort="Klein, George J" uniqKey="Klein G" first="George J." last="Klein">George J. Klein</name><name sortKey="Krahn, Andrew D" sort="Krahn, Andrew D" uniqKey="Krahn A" first="Andrew D." last="Krahn">Andrew D. Krahn</name><name sortKey="Mohamed, Uwais" sort="Mohamed, Uwais" uniqKey="Mohamed U" first="Uwais" last="Mohamed">Uwais Mohamed</name><name sortKey="Petrellis, Basilios" sort="Petrellis, Basilios" uniqKey="Petrellis B" first="Basilios" last="Petrellis">Basilios Petrellis</name><name sortKey="Redfearn, Damian P" sort="Redfearn, Damian P" uniqKey="Redfearn D" first="Damian P." last="Redfearn">Damian P. Redfearn</name><name sortKey="Skanes, Allan C" sort="Skanes, Allan C" uniqKey="Skanes A" first="Allan C." last="Skanes">Allan C. Skanes</name><name sortKey="Yee, Raymond" sort="Yee, Raymond" uniqKey="Yee R" first="Raymond" last="Yee">Raymond Yee</name></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
EXPLOR_STEP=$WICRI_ROOT/Wicri/Canada/explor/ParkinsonCanadaV1/Data/Main/Exploration
HfdSelect -h $EXPLOR_STEP/biblio.hfd -nk 002867 | SxmlIndent | more
Ou
HfdSelect -h $EXPLOR_AREA/Data/Main/Exploration/biblio.hfd -nk 002867 | SxmlIndent | more
Pour mettre un lien sur cette page dans le réseau Wicri
{{Explor lien
|wiki= Wicri/Canada
|area= ParkinsonCanadaV1
|flux= Main
|étape= Exploration
|type= RBID
|clé= ISTEX:B48FB2053E9197FF284325910055B400156E0503
|texte= Characteristics of Slow Pathway Conduction After Successful AVNRT Ablation
}}
| This area was generated with Dilib version V0.6.29. |  |